Our main idea is that RNAi can be used to selectively reduce the production of mutant huntingtin is slow or block the progression of HD, but it is also assumed that the excessive neural function can inhibit huntingtin gene silencing by interfering in the most important signaling events," said Aronia Neil, MD, UMMS professor of medicine and cell biology and principal investigator of the study."This research collaboration will allow us to identify promising RNAi-based therapeutic action of selective mutant huntingtin test vehicles with the aim of restoring normal nerve function. We have a long way to push this research forward, and this is the next step Lundbeck very exciting. "
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